Myocardial Ischemia and Preconditioning
Naranjan S. Dhalla Β· Nobuakira Takeda Β· Manjeet Singh Β· Anton Lukas
Π΄Π΅ΠΊ. 2012β―Π³. Β· Progress in Experimental Cardiology ΠΠ½ΠΈΠ³Π° 6 Β· Springer Science & Business Media
Π-ΠΊΠ½ΠΈΠ³Π°
541
Π‘ΡΡΠ°Π½ΠΈΡΠΈ
reportΠΡΠ΅Π½ΠΈΡΠ΅ ΠΈ ΡΠ΅ΡΠ΅Π½Π·ΠΈΠΈΡΠ΅ Π½Π΅ ΡΠ΅ ΠΏΠΎΡΠ²ΡΠ΄Π΅Π½ΠΈ Β ΠΠΎΠ·Π½Π°ΡΡΠ΅ ΠΏΠΎΠ²Π΅ΡΠ΅
ΠΠ° Π΅-ΠΊΠ½ΠΈΠ³Π°Π²Π°
Current evidence suggests that the ischemic preconditioning response is a multi-factorial process consisting of an initial early trigger, an intermediate mediator, and an end effector. Each of these steps in is now its own intense area of investigation. The need to render the heart ischemic for a brief period to invoke the preconditioning response is currently the major factor limiting clinical application of this powerful cardioprotective strategy. Recent research efforts have utilized brief exposures to pharmacological agents, in lieu of a brief preconditioning ischemia, to trigger/mimic the ischemic preconditioning-induced response. The World Heart Congress held in Winnipeg in July 2001 provided a forum for the presentation of new insights into the basic mechanisms of ischemia and reperfusion injury, as well as novel strategies to protect the heart from cell death, ventricular arrhythmias, and contractile dysfunction. Many pioneers in the fields of ischemia-reperfusion injury and preconditioning-induced protection presented there and the chapters in this book represent selected papers from these symposia.
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ΠΠ° Π΄Π° ΡΠΈΡΠ°ΡΠ΅ Π½Π° ΡΡΠ΅Π΄ΠΈ ΡΠΎ Π΅-ΠΌΠ°ΡΡΠΈΠ»ΠΎ, ΠΊΠ°ΠΊΠΎ ΡΡΠΎ ΡΠ΅ Π΅-ΡΠΈΡΠ°ΡΠΈΡΠ΅ Kobo, ΡΠ΅ ΡΡΠ΅Π±Π° Π΄Π° ΠΏΡΠ΅Π·Π΅ΠΌΠ΅ΡΠ΅ Π΄Π°ΡΠΎΡΠ΅ΠΊΠ° ΠΈ Π΄Π° ΡΠ° ΠΏΡΠ΅ΡΡΠ»ΠΈΡΠ΅ Π½Π° ΡΡΠ΅Π΄ΠΎΡ. Π‘Π»Π΅Π΄Π΅ΡΠ΅ Π³ΠΈ Π΄Π΅ΡΠ°Π»Π½ΠΈΡΠ΅ ΡΠΏΠ°ΡΡΡΠ²Π° Π²ΠΎ Π¦Π΅Π½ΡΠ°ΡΠΎΡ Π·Π° ΠΏΠΎΠΌΠΎΡ Π·Π° ΠΏΡΠ΅ΡΡΠ»Π°ΡΠ΅ Π½Π° Π΄Π°ΡΠΎΡΠ΅ΠΊΠΈΡΠ΅ Π½Π° ΠΏΠΎΠ΄Π΄ΡΠΆΠ°Π½ΠΈ Π΅-ΡΠΈΡΠ°ΡΠΈ.
